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Inhibition of host N-myristoylation compromises the infectivity of SARS-CoV-2 due to Golgi-bypassing egress

  • Saber H. Saber
  • , Nyakuoy Yak
  • , Konstantin Dolski
  • , Sanna Mäki
  • , Lev Levanov
  • , Levina A. Willenbrink
  • , Julian D.J. Sng
  • , Mohammed R. Shaker
  • , Sean D. Morrison
  • , Huiwen Zheng
  • , Selin Pars
  • , Giovanni Pietrogrande
  • , Yih Tyng Bong
  • , Tania Vane-Tempest
  • , Teemu Smura
  • , Tomas Strandin
  • , Ravi Ojha
  • , Ravi Kant
  • , Janika Ruuska
  • , Francesco Topi
  • Diana Vaskiv, Lauri Kareinen, Tobias Binder, Siyuan Lu, Matthias Floetenmeyer, Bahaa Al-mhanawi, Yanshan Zhu, Tarja Sironen, Gert Hoy Talbo, Kirsty R. Short, Wouter W. Kallemeijn, Roberto Solari, Jessica Mar, Edward W. Tate, Ashley J. van Waardenberg, Olli Vapalahti, Ernst Wolvetang*, Giuseppe Balistreri*, Merja Joensuu*
*Corresponding author for this work
  • University of Queensland
  • Assiut University
  • University of Helsinki
  • Medical University of Gdańsk
  • Finnish Food Authority
  • Francis Crick Institute
  • Imperial College London
  • Myricx Bio
  • i-Synapse PTY LTD
  • Helsinki University Hospital

Research output: Contribution to journalArticlepeer-review

Abstract

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), which caused the coronavirus disease 2019 (COVID-19) pandemic, remains a global health concern despite vaccines, neutralizing antibodies, and antiviral drugs. The emergence of viral mutations that diminish the effectiveness of current interventions underscores the importance of alternative, host-directed strategies. Here, we show that pharmacological inhibition or knockdown of host N-myristoyltransferase 1 (NMT1), one of the two human enzymes that mediates protein N-myristoylation, significantly impairs SARS-CoV-2, Vesicular Stomatitis Virus (VSV) and Respiratory syncytial virus (RSV) infections. We demonstrate the antiviral efficacy and safety of this host-directed therapeutic strategy across multiple viral tropic sites, including human lung adenocarcinoma cell lines, primary nasal epithelial cells, and human choroid plexus-cortical brain organoids. NMT1 inhibition triggers a Golgi-bypassing pathway for SARS-CoV-2 progeny virion egress, through endoplasmic reticulum and lysosomal structures, which leads to perturbed progeny virion composition and spike maturation, impairing progeny virion infectivity.

Original languageEnglish
Article number7055
JournalNature Communications
Volume17
Issue number1
DOIs
Publication statusPublished - Dec 2026
Externally publishedYes

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